Carcinogenesis and Cancer

Carcinogenesis is the creation of a cancer.ceases to divide. Tumor cells keep dividing forever.
Cancer is, ultimately, a disease of genes. Typically, aAltered morphology.
series of several mutations is required before a cellBuilding of cellular clusters (Foci).
becomes a cancer cell. We distinguish betweenLoss of contact inhibition.
oncogenes, which promote cancer when "switchedLow or no need for growth factors.
on" by a mutation, and tumor suppressor genes,Metastasis
which prevent cancer unless "switched off" by aCancers are capable of spreading through the body
mutation. These mutations can have various causes:by two mechanisms: local invasion and distant
radiation or chemicals called carcinogens; somemetastasis. Invasion refers to the direct migration
inherited predisposition is not uncommon; someand penetration by cancer cells into neighboring
viruses that can cause cancer have also beentissues. Metastasis refers to the ability of cancer cells
described. Usually, they carry in their genome someto penetrate into lymphatic and blood vessels,
oncogene or tumor suppressor inactivating gene. Incirculate through the bloodstream, and then invade
about 15% of all cancers, viruses seem to play a role.normal tissues elsewhere in the body. Cancer is most
Finally, damage by free radicals, which are a naturaldeadly when it metastasizes.
by-product of oxygen metabolism, can causeForms of cancer
mutations in the DNA.The term cancer is very broad and covers many
For most of the cancers, it cannot be told whichdifferent illnesses including:
event was the initial cause. However, with molecularBladder cancer
biology, it is possible to characterize the mutationsBone cancer
within a tumor, and to a certain extent to predict itsBrain tumor
behavior. For example, about half of the tumors areBreast cancer
deficient in the tumor suppressor gene p53, alsoCervical cancer
known as "the guardian of the genome". This isColorectal cancer (includes colon, rectum, anus, and
associated with poor prospects for the patient, sinceappendix)
those tumor cells are unlikely to go into apoptosisCancer of the esophagus
(programmed cell death) after they are damaged byHodgkin's disease
therapy. There are more mutations that make aKidney cancer
tumor more malignant. Telomerase mutations enableCancer of the larynx
a tumor cell to divide indefinitely. Other mutationsLeukemia
enable the tumor to grow new blood vessels to feedLiver cancer
it, or to detach from the surrounding tissue,Lung cancer
spreading to other parts of the body.Lymphoma
Malignant tumors such as carcinoma or sarcoma,Melanoma
lymphoma or leukemia originate from a cell or a groupMoles and dysplastic nevi
of cells in a multicellular organism that have severalMultiple myeloma
distinct properties:increased cell division ratenotMuscular Cancer
controllable by growth factors anymorealteredNon-Hodgkin's lymphoma
differentiation (specialization) abilityno ability forOral cancer
contact inhibitionability to invade neighbouringOvarian cancer
tissueability to build metastasesability to promoteCancer of the pancreas
blood vessel growthProstate cancer
A cell that degenerates into a tumor cell does usuallySkin cancer
not acquire all these properties at once, but itsStomach cancer
daughter cells are selected to build them. ThisTesticular cancer
process is called cellular evolution. A first step in theTeratoma
development of a tumor cell is usually a small changeThyroid cancer
in the DNA, often a point mutation, which leads,Cancer of the uterus
among other things, to a genetic instability of the cell.Clinical aspects
The instability increases to a point where the cellThe field of medicine concerned with the treatment
loses whole chromosomes, or has double ones. Also,of cancer is oncology.
the DNA methylation pattern of the cell changes,Cancer has become an important problem with the
activating and deactivating genes more or less atrise in life expectancy, as the above mentioned
random. Cells that divide at a high rate, such as stemmutations become more likely the longer a person
cells, show a higher risk of becoming tumor cells thanlives. Though great progress in treatment has been
those which divide less or not at all, for example,made, most cancers in advanced stages remain
neurons. If the initial tumor cell (or group of tumorincurable and ultimately fatal.
cells) is not removed by the immune system, it willTreatment of cancer typically involves surgery to
develop into cancer.remove tumors and nearby lymph nodes to which
In cellular model systems, cells are exposed tothe cancer may have spread, combined with radiation
carcinogenic influences (chemicals, radiation). In thesetherapy and/or chemotherapy. The latter two target
systems, the first signs of a cell developing into acells in the body that are rapidly dividing. This includes
tumor cell are :the cancer cells but also certain healthy ones, which is
Immortality. The usual number of cell divisions for athe reason for the severe side effects of these
mammalian cell is 50-60 (cell senescence), then ittreatments.