The truth and how to prevent cancer


Can it be prevented?

Cancer prevention is defined as activehave been validated in an observational
measures to decrease the incidence of(or occasionally a prospective
cancer. This can be accomplished byinterventional) trial in humans.
avoiding carcinogens or altering theirThe case of beta-carotene provides an
metabolism, pursuing a lifestyle or dietexample of the necessity of randomized
that modifies cancer-causing factors andclinical trials. Epidemiologists
or medical interventionstudying both diet and serum levels
(chemoprevention, treatment ofobserved that high levels of
pre-malignant lesions).beta-carotene, a precursor to vitamin A,
Much of the promise for cancerwere associated with a protective
prevention comes from observationaleffect, reducing the risk of cancer.
epidemiologic studies that showThis effect was particularly strong in
associations between modifiable lifelung cancer. This hypothesis led to a
style factors or environmental exposuresseries of large randomized trials
and specific cancers. Evidence is nowconducted in both Finland and the United
emerging from randomized controlledStates (CARET study) during the 1980s
trials designed to test whetherand 1990s. This study provided about
interventions suggested by the80,000 smokers or former smokers with
epidemiologic studies, as well as leadsdaily supplements of beta-carotene or
based on laboratory research, actuallyplacebos. Contrary to expectation, these
result in reduced cancer incidence andtests found no benefit of beta-carotene
mortality.supplementation in reducing lung cancer
Examples of modifiable cancer riskincidence and mortality. In fact, the
include alcohol consumption (associatedrisk of lung cancer was slightly, but
with increased risk of oral, esophageal,not significantly, increased by
breast, and other cancers), smokingbeta-carotene, leading to an early
(although 20% of women with lung cancertermination of the study. [10]
have never smoked, versus 10% of menHowever, Randomized Clinical Trials
[1]), physical inactivity (associated(RCTs) also have drawbacks in cancer
with increased risk of colon, breast,prevention, particularly in
and possibly other cancers), and beingmicronutrient deficiencies, which are
overweight (associated with colon,thought by some to be a major
breast, endometrial, and possibly othercontributor to cancer. RCTs involve huge
cancers). Based on epidemiologicnumbers of people, take many years to
evidence, it is now thought thatcomplete, and are therefore extremely
avoiding excessive alcohol consumption,expensive and complicated, and therefore
being physically active, and maintainingfew are done. In addition, these
recommended body weight may allrandomized clinical trials usually test
contribute to reductions in risk ofonly a single dose. An alternative,
certain cancers; however, compared withwhich is likely to be more useful, is to
tobacco exposure, the magnitude ofdo shorter intervention trials focusing
effect is modest or small and theon other endpoints related to cancer,
strength of evidence is often weaker.such as DNA damage. These trials can
Other lifestyle and environmentaltest a variety of doses on fewer people
factors known to affect cancer riskto determine what level of micronutrient
(either beneficially or detrimentally)intake (or, better, micronutrient
include certain sexual and reproductiveconcentration in blood) keeps DNA damage
practices [citation needed], the use ofto a minimum.
exogenous hormones, exposure to ionizingOther chemoprevention agents
radiation and ultraviolet radiation,Daily use of tamoxifen, a selective
certain occupational and chemicalestrogen receptor modulator, typically
exposures, and infectious agents.for 5 years, has been demonstrated to
Diet and cancerreduce the risk of developing breast
The consensus on diet and cancer is thatcancer in high-risk women by about 50%.
obesity increases the risk of developingCis-retinoic acid also has been shown to
cancer. Particular dietary practicesreduce risk of second primary tumors
often explain differences in canceramong patients with primary head and
incidence in different countries (e.g.neck cancer. Finasteride, a 5-alpha
gastric cancer is more common in Japan,reductase inhibitor, has been shown to
while colon cancer is more common in thelower the risk of prostate cancer. Other
United States). Studies have shown thatexamples of drugs that show promise for
immigrants develop the risk of their newchemoprevention include COX-2 inhibitors
country, suggesting a link between diet(which inhibit a cyclooxygenase enzyme
and cancer rather than a genetic basis.involved in the synthesis of
Despite frequent reports of particularproinflammatory prostaglandins).
substances (including foods) having aGenetic testing
beneficial or detrimental effect onGenetic testing for high-risk
cancer risk, few of these have anindividuals, with enhanced surveillance,
established link to cancer. Thesechemoprevention, or risk-reducing
reports are often based on studies insurgery for those who test positive, is
cultured cell media or animals. Publicalready available for certain
health recommendations cannot be made oncancer-related genetic mutations.
the basis of these studies until they



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